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Abstract Left ventricular hypertrophy (LVH) is a major cardiovascular complication of chronic kidney disease (CKD), yet its mechanisms remain poorly understood. Here we show that in preclinical experiments, CKD drives cardiomyocyte metabolic reprogramming characterized by enhanced glutaminolysis and impaired oxidative phosphorylation in male C57BL/6J mice.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
