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ABSTRACT Ebola virus enters host cells via the viral glycoprotein binding to host Niemann-Pick disease, type C1 protein C domain (NPC1-C), initiating membrane fusion. Our research focused on enhancing drug development targeting NPC1 to combat fatal Ebola virus-caused haemorrhagic fever.Improving existing drugs with high NPC1 affinity and generating novel compounds through bioisosteric modelling are two drug-designing strategies.
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