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Abstract Type 1 and type 2 diabetes are characterized by beta cell dysfunction and insulin deficiency, and the appropriate turnover of MYC expression appears to control the beta-cell fate. Therefore, it is important to better understand the mechanism that controls MYC expression. Hence, the current study aimed to elucidate the interactions of the long non-coding RNA molecule PHAROH at the interface of MYC mRNA and the RNA-binding protein TIA1.
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