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Sladjana Skopelja-Gardner, Lucrezia Colonna, Payton Hermanson, Xizhang Sun, Lena Tanaka, Joyce Tai, Yenly Nguyen, Jessica M. Snyder, Charles E. Alpers, Kelly L. Hudkins, David J. Salant, YuFeng Peng and Keith B. Elkon J Immunol May 15, 2020, 204 (10) 2627-2640; DOI: https://doi.org/10.4049/jimmunol.1901473 Key Points Novel TM lupus mouse strains develop spontaneous nephritis. In C1q deficiency, kidney complement activation likely occurred via the LP.
Sladjana Skopelja-Gardner, Lucrezia Colonna, Payton Hermanson, Xizhang Sun, Lena Tanaka, Joyce Tai, Yenly Nguyen, Jessica M. Snyder, Charles E. Alpers, Kelly L. Hudkins, David J. Salant, YuFeng Peng and Keith B. Elkon J Immunol April 1, 2020, ji1901473; DOI: https://doi.org/10.4049/jimmunol.1901473 Key Points Novel TM lupus mouse strains develop spontaneous nephritis. In C1q deficiency, kidney complement activation likely occurred via the LP.
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