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Abstract Competitive victimhood denotes group members’ efforts to establish that their ingroup has suffered greater injustice than an adversarial outgroup. Previous research in contexts of structural inequality has stressed the role of the need to defend the ingroup's moral identity, rather than the need for power, in leading advantaged and disadvantaged group members to engage in competitive victimhood.
Focal segmental glomerulosclerosis (FSGS) is a scarring process associated with chronic low‐grade inflammation ascribed to toll‐like receptor (TLR) activation and monocyte migration. We developed synthetic, small‐molecule lecinoxoids, VB‐201 and VB‐703, that differentially inhibit TLR‐2‐ and TLR‐4‐mediated activation and monocyte migration. The efficacy of anti‐inflammatory lecinoxoid treatment on FSGS development was explored using a 5/6 nephrectomy rat model.
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