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Abstract Osteoclast bone resorption consumes a large amount of energy, and its energy metabolism disorder is an important pathogenesis of bone metabolism diseases such as osteoporosis. Excessive copper ions interfere with the energy metabolism of the mitochondrial tricarboxylic acid cycle, leading to cuproptosis—a recently identified form of regulated cell death. Ceruloplasmin (CP), a major copper-binding protein, is associated with osteoporosis.
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