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Abstract Maternal immune activation (MIA) can persistently alter offspring neuroimmune functions, but its contribution to the susceptibility to neurodegenerative diseases remains unclear. We tested whether Poly(I:C)-induced MIA enhances a latent vulnerability to mechanistically distinct PD-related insults. Single-cell RNA sequencing of adult offspring's brain revealed broadly preserved cellular composition but identified a disease-relevant transcriptional signature centered on astrocytes.
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