Renda Li
As seen in:
Nature,
Wiley Online Library,
MDPI,
JAMA Oncology,
Journal of Nuclear Medicine,
Lung Cancer
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Articles by Renda Li
Development of Dar3A Chelator and Construction of FAP-Targeted PET Probe as Proof of Concept and for First-in-Human Evaluation
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Development of Dar3A Chelator and Construction of FAP-Targeted PET Probe as Proof of Concept and for First-in-Human Evaluation
Thank you for your interest in spreading the word on Journal of Nuclear Medicine. NOTE: We only request your email address so that the person you are recommending the page to knows that you wanted them to see it, and that it is not junk mail. We do not capture any email address.
Lymph Node Dissection Guideline Adherence and Survival in Patients With T1N0M0 Lung Adenocarcinoma
Supplement 1. eFigure 1. Diagram of the study population selection steps eFigure 2. Survival curves by histologic pattern group of lung adenocarcinoma eFigure 3. Survival curves by compliance to lymph node dissection guideline eFigure 4. Directed acyclic graph eFigure 5. Absolute standardized mean difference before and after weighting eFigure 6. Histograms of stabilized weights Supplement 2. eTable 1.
[2504.00870] Data-free Knowledge Distillation with Diffusion Models
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Identifying the Hierarchical Structure of Nighttime Economic Agglomerations Based on the Fusion of Multisource Data
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Development and validation of a nomogram based on preoperative variables for predicting recurrence‐free survival in stage IA lung adenocarcinoma
INTRODUCTION Lung cancer is one of the most frequently diagnosed cancers and the leading cause of cancer-related deaths worldwide, with an estimated 2 million new cases and 1.76 million deaths per year.1 There are two main types of primary lung cancer: non-small cell lung cancer (NSCLC) and small cell lung cancer (SCLC).
Integrated molecular characterization reveals potential therapeutic strategies for pulmonary sarcomatoid carcinoma
Abstract Pulmonary sarcomatoid carcinoma (PSC) is a rare subtype of lung cancer with poor prognosis. Here, we perform multi-omics analysis of 56 PSC samples, 14 of which are microdissected to analyze intratumoral heterogeneity. We report the mutational landscape of PSC. The epithelial and sarcomatoid components share numerous genomic alterations, indicating a common progenitor. We find that epithelial-mesenchymal transition (EMT) plays important roles in the carcinogenesis of PSC.
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