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Articles by Roisin Sullivan
Vibrio harveyi plasmids as drivers of virulence in barramundi (Lates calcarifer)?
Abstract Vibrio species are an emerging public and animal health risk in marine environments and the opportunistic bacterial pathogen Vibrio harveyi is a major disease risk for tropical aquaculture. Current understanding of virulence in V. harveyi is limited by strain-specific variability and complex host-pathogen dynamics.
Replacement of fishmeal with a microbial single‐cell protein induced enteropathy and poor growth outcomes in barramundi (Lates calcarifer) fry
1 INTRODUCTION The increasing global demand for protein is a direct response to the challenge of sustainably feeding the projected world population of 9.7 billion by 2050 (United Nations, 2023). Despite the adoption of the UN Sustainable Development Goals (SDG) in 2015, including SDG 2 (Zero Hunger), global undernourishment has risen to 9.3% in 2021 (FAO, IFAD, UNICEF, WFP and WHO, 2022).
RFC1 repeat expansion analysis from whole genome sequencing data simplifies screening and increases diagnostic rates
The authors have declared no competing interest. This research was made possible through access to the data and findings generated by the 100,000 Genomes Project. The 100,000 Genomes Project is managed by Genomics England Limited (a wholly owned company of the Department of Health and Social Care). The 100,000 Genomes Project is funded by the National Institute for Health Research and NHS England.
An optimised saliva collection method to produce high-yield, high-quality RNA for translational research
Loading metrics Open Access Peer-reviewed Research Article Roisin Sullivan, Susan Heavey, David G. Graham, Rachel Wellman, Saif Khan, Sri Thrumurthy, Benjamin S. Simpson, Tina Baker, Sarah Jevons, Jose Ariza, Victor Eneh, Hayley Pye, Hayley Luxton, Rifat Hamoudi, Hayley Whitaker , [ ... ], Laurence B. Lovat [ view all ] [ view less ] Roisin Sullivan, Susan Heavey, David G. Graham, Rachel Wellman, Saif Khan, Sri Thrumurthy, Benjamin S.
Author Correction: Biallelic expansion of an intronic repeat in RFC1 is a common cause of late-onset ataxia
Correction to: Nature Genetics https://doi.org/10.1038/s41588-019-0372-4, published online 29 March 2019. In the version of this article initially published, the name of author Wai Yan Yau was misspelled. The error has been corrected in the HTML and PDF versions of the article. Author information Author notes These authors contributed equally: Roisin Sullivan, Jana Vandrovcova. These authors jointly supervised this project: Mary M. Reilly, Henry Houlden.
Biallelic expansion of an intronic repeat in RFC1 is a common cause of late-onset ataxia
Abstract Late-onset ataxia is common, often idiopathic, and can result from cerebellar, proprioceptive, or vestibular impairment; when in combination, it is also termed cerebellar ataxia, neuropathy, vestibular areflexia syndrome (CANVAS).
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