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Interleukin (IL)-17 plays a central role in the immunopathogenesis of psoriasis.1,2 This cytokine family, which includes several structurally related proteins designated IL-17A to IL-17F, stimulates T-cell-mediated inflammation and keratinocyte proliferation and helps drive the psoriasis inflammatory cascade.1–3 Blocking IL-17 activity results in a rapid reduction in psoriatic disease severity.3 Most anti-IL-17 agents in clinical use are monoclonal antibodies.
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