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As seen in: Frontiers in Immunology, JACC: Cardiovascular Interventions
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Articles by Run Du

Glymphatic surrogate, choroid plexus remodeling, and exploratory brain network analysis in the post-acute phase of anti-NMDAR encephalitis: multimodal MRI evidence for cognitive associations and differential white matter mediation

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By Run Du, Jinyu Tao, Yayun Xiang, Qiyuan Zhu, Zhuowei Shi, Wenwen Wang, Yongmei Li, Chun Zeng, Junwei Xiong
| Frontiers in Immunology
Abstract Background: Anti-N-methyl-D-aspartate receptor (anti-NMDAR) encephalitis typically presents with acute neuroinflammation, and many patients often experience persistent cognitive impairment during the post-acute/recovery phase. While impaired cerebrospinal fluid (CSF)-related markers—including glymphatic function and choroid plexus (CP) structural remodeling—are well documented in neurodegenerative diseases, their role in acute autoimmune brain injury remains unclear.
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Minimizing Permanent Pacemaker Following Repositionable Self-Expanding Transcatheter Aortic Valve Replacement

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By Hasan Jilaihawi, Zhengang Zhao, Run Du, Cezar Staniloae
| JACC: Cardiovascular Interventions Verified
JACC: Cardiovascular Interventions Focus on Complex Transcatheter Aortic Valve Replacement Cases Hasan Jilaihawi, Zhengang Zhao, Run Du, Cezar Staniloae, Muhamed Saric, Peter J. Neuburger, Michael Querijero, Alan Vainrib, Kazuhiro Hisamoto, Homam Ibrahim, Tara Collins, Emily Clark, Illya Pushkar, Daniel Bamira, Ricardo Benenstein, Afnan Tariq and Mathew Williams This article requires a subscription or purchase to view the full text.
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Minimizing Permanent Pacemaker Following Repositionable Self-Expanding Transcatheter Aortic Valve Replacement

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By Hasan Jilaihawi, Zhengang Zhao, Run Du, Cezar Staniloae
| jacc.org
JACC: Cardiovascular Interventions Clinical Research Hasan Jilaihawi, Zhengang Zhao, Run Du, Cezar Staniloae, Muhamed Saric, Peter J. Neuburger, Michael Querijero, Alan Vainrib, Kazuhiro Hisamoto, Homam Ibrahim, Tara Collins, Emily Clark, Illya Pushkar, Daniel Bamira, Ricardo Benenstein, Afnan Tariq and Mathew Williams This article requires a subscription or purchase to view the full text.
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p‐Cresyl Sulfate Aggravates Cardiac Dysfunction Associated With Chronic Kidney Disease by Enhancing Apoptosis of Cardiomyocytes

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By Hui Han, Jinzhou Zhu, Zhengbin Zhu, Jingwei Ni, Run Du, Yang Dai, Yanjia Chen, Zhijun Wu, Lin Lu
| jaha.ahajournals.org
Background Cardiovascular disease is the leading cause of death in patients with chronic kidney disease. A body of evidence suggests that p‐cresyl sulfate (PCS), a uremic toxin, is associated with the cardiovascular mortality rate of patients with chronic kidney disease; however, the molecular mechanisms underlying this feature have not yet been fully elucidated.
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