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Abstract EphA2, a member of Eph family receptor tyrosine kinases (RTKs), is overexpressed in multiple types of solid human tumors, particularly at the late stages. However, whether and how it drives specific malignant processes remain elusive. We report that EphA2 is elevated during prostate cancer (PCa) progression in multiple syngeneic murine models.
Abstract Eph receptors are the largest family of receptor tyrosine kinases (RTKs). They play a role in the pathogenesis of various diseases including cancer, atherosclerosis, fibrosis, infectious diseases, diseases of the central nervous system and age-related cataract. EphA2 has attracted much attention over the years owing to its dysregulation in many diseases.
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