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1 Introduction Heart failure (HF) is pathological structural myocardial remodelling including cardiomyocyte hypertrophy, fibrosis, cardiomyocyte cell death and fibrosis. Chronic, low-grade inflammation is also one of the major factors implicated in the development and progression of HF [1-3]. Polymorphonuclear neutrophils are terminally differentiated cells and are the most abundant subset of leukocytes and the major constituents of the innate immune system.
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