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Articles by Side Liu
Sa1171 CLINICAL EVALUATION OF A SERUM 12-MIRNA TEST FOR GASTRIC CANCER SCREENING IN A HIGH-RISK CHINESE POPULATION: A PROSPECTIVE MULTICENTRE STUDY
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Publisher Correction: RCN3 functions as a tumor promoter in colorectal cancer by modulating the GRP78-PI3K-AKT signaling pathway - Oncogene
Correction to: Oncogene https://doi.org/10.1038/s41388-025-03535-5, published online 23 August 2025 Following the publication of this article, it was noted that incorrect images had been published for Figures 6j and k. Figures 6l and m had mistakenly been duplicated by the Publisher during typesetting. Figures 6j and k have now been replaced with the original figures provided. The original article has been corrected. About this article Huang, M., Qin, B., Xie, Z. et al.
RCN3 functions as a tumor promoter in colorectal cancer by modulating the GRP78-PI3K-AKT signaling pathway - Oncogene
Abstract Colorectal cancer (CRC) remains a significant global health concern, highlighting the urgent need for novel therapeutic targets and prognostic biomarkers. This study investigates the role of Reticulocalbin-3 (RCN3) in CRC development, focusing on its regulatory influence on calcium ion homeostasis and transport proteins, critical factors in cancer progression.
ARID1A loss enhances sensitivity to c-MET inhibition by dual targeting of GPX4 and iron homeostasis, inducing ferroptosis - Cell Death & Differentiation
Abstract ARID1A, a subunit of the SWI/SNF chromatin-remodeling complex, functions as a tumor suppressor in various cancer types. Owing to its high frequency of inactivating mutations, ARID1A has emerged as a promising target for the development of anticancer drugs. In this study, we report that ARID1A-deficient colorectal cancer (CRC) cells induce synthetic lethality when treated with inhibitors of c-MET receptor tyrosine kinase.
Long non-coding RNA-MIR181A1HG acts as an oncogene and contributes to invasion and metastasis in gastric cancer - Oncogene
Abstract Dysregulation of long non-coding RNAs (lncRNA) plays an essential role in cancer development and progression. However, their functions and mechanisms of action in gastric cancer (GC) remain largely unknown. Gene expression in GC was evaluated using quantitative real-time PCR, western blotting, immunofluorescence, immunohistochemistry, and RNA in situ hybridization.
Therapeutic targeting of ARID1A-deficient cancer cells with RITA (Reactivating p53 and inducing tumor apoptosis) - Cell Death & Disease
Abstract ARID1A, a component of the SWI/SNF chromatin-remodeling complex, is frequently mutated in various cancer types and has emerged as a potential therapeutic target. In this study, we observed that ARID1A-deficient colorectal cancer (CRC) cells showed synthetic lethal effects with a p53 activator, RITA (reactivating p53 and inducing tumor apoptosis). RITA, an inhibitor of the p53-MDM2 interaction, exhibits increased sensitivity in ARID1A-deficient cells compared to ARID1A wild-type cells.
The VAX2-LINC01189-hnRNPF signaling axis regulates cell invasion and migration in gastric cancer - Cell Death Discovery
Abstract Transcription factors (TFs) and long noncoding RNAs (lncRNAs) contribute to gastric cancer (GC). However, the roles of TFs and lncRNAs in the invasion and metastasis of GC remain largely unknown. Here, we observed that the transcription factor VAX2 is significantly upregulated in GC cells and tissues and acts as an oncogene. Moreover, high VAX2 expression is associated with the advancement of tumors in GC.
The HOXD9-mediated PAXIP1-AS1 regulates gastric cancer progression through PABPC1/PAK1 modulation - Cell Death & Disease
Abstract Long non-coding RNAs (lncRNAs) have been functionally characterised in various diseases. LncRNA PAX-interacting protein 1-antisense RNA 1 (PAXIP1-AS1) has reportedly been associated with cancer development. However, its role in gastric cancer (GC) remains poorly understood. Here, we showed that PAXIP1-AS1 was transcriptionally repressed by homeobox D9 (HOXD9) and was significantly downregulated in GC tissues and cells.
METTL14-dependent maturation of pri-miR-17 regulates mitochondrial homeostasis and induces chemoresistance in colorectal cancer - Cell Death & Disease
Abstract miR-17-5p has been found to be involved in the proliferation and metastasis of colorectal cancer (CRC), and N6-methyladenosine (m6A) modification is the most common RNA modification in eukaryotes. However, whether miR-17-5p contributes to chemotherapy sensitivity in CRC via m6A modification is unclear.
Prognostic and predictive value of a pathomics signature in gastric cancer - Nature Communications
Abstract The current tumour-node-metastasis (TNM) staging system alone cannot provide adequate information for prognosis and adjuvant chemotherapy benefits in patients with gastric cancer (GC). Pathomics, which is based on the development of digital pathology, is an emerging field that might improve clinical management. Herein, we propose a pathomics signature (PSGC) that is derived from multiple pathomics features of haematoxylin and eosin-stained slides.
The POU2F1/miR-4490/USP22 axis regulates cell proliferation and metastasis in gastric cancer
Comments Cellular Oncology (2020) - doi: 10.1007/s13402-020-00553-1 issn: 2211-3428 issn: 2211-3436 pubmed: 32857323 Yizhi Xiao, Side Liu, Jiaying Li, Weiyu Dai, Weimei Tang, Li Xiang, Wenjing Zhang, Jianjiao Lin, Jing Wang, Xiaosheng Wu, Guangnan Liu, Yuyang Liu, Yaying Chen, Huiqiong Zhu, Yusi Wang, Zhizhao Lin, Qiong Yang, Tianming Chen, Yong Sun, Aimin Li
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