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Abstract DNA polymerase theta (Pol θ)is a conserved an A-family polymerase that plays an essential role in repairing double strand breaks, through micro-homology end joining, and bypassing DNA lesions, through translesion synthesis, to protect genome integrity. Despite its essential role in DNA repair, Pol θ is inherently error prone. Recently, Pol θ has been identified in various cancer types, suggesting Pol θ's promiscuous nature aids in cancer progression.
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