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Atopic dermatitis (AD) prevalence has been increasing over the past 20 years, affecting infants but also occurring or persisting into adulthood.1 The main objective of the PreDDA study was to estimate adult prevalence of AD in a French representative population-based cohort of adults (CONSTANCES),2 and to describe factors associated with different profiles of AD.
Abstract A dysfunctional epidermal barrier, which may be associated with mutations in the filaggrin gene in genetically predisposed individuals or harmful effects of environmental agents and allergens, contributes to the development of atopic dermatitis (AD) due to an interplay between the epithelial barrier, immune defence and the cutaneous microbiome.
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