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Dear Editor, We read with great interest the study by Kmieć et al. [1] examining whether overnight dexamethasone suppression can improve the interpretability of aldosterone-to-renin ratio (ARR)-based screening for primary aldosteronism in patients receiving renin–angiotensin–aldosterone system–interfering therapy. The protocol addresses a clinically relevant diagnostic barrier that continues to limit the detection of a common and treatable cause of hypertension.
Introduction Alzheimer’s disease (AD) is a multifactorial neuro-degenerative disease, which affects cognitive function and memory, is characterized by amyloid β (Aβ, formed by activities of β and γ secretase) and neurofibrillary tangles (NFTs, composed of over-phosphorylated tau protein), but its pathogenesis is not yet clear (Blennow and Zetterberg, 2018; Weller and Budson, 2018).
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