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Abstract B cells are central mediators of humoral immunity, and their development is tightly regulated to prevent autoreactivity. The unfolded protein response (UPR) plays a crucial role in supporting the high biosynthetic demands of B cells. The transcription factor XBP1s, a key effector of the UPR’s IRE1α branch, has been implicated in plasma cell differentiation, but its role in early B cell development and tolerance remains unclear.
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