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Articles by Takao Urabe
Survivin Overexpression via Adeno‐Associated Virus Vector Rh10 Ameliorates Ischemic Damage after Middle Cerebral Artery Occlusion in Rats
Survivin, a member of the inhibitors of apoptosis protein gene family, inhibits the activity of caspase, leading to a halt of the apoptotic process. Our study focused on the neuroprotective effect of survivin after transient middle cerebral artery occlusion (MCAO) with intraparenchymal administration of an adeno‐associated virus (AAV) vector. His‐tagged survivin was cloned and packaged into the AAV‐rh10 vector.
Survivin overexpression via adeno‐associated virus vector Rh10 ameliorates ischemic damage after middle cerebral artery occlusion in rats
Survivin, a member of the inhibitors of apoptosis protein gene family, inhibits the activity of caspase, leading to a halt of the apoptotic process. Our study focused on the neuroprotective effect of survivin after transient middle cerebral artery occlusion (MCAO) with intraparenchymal administration of an adeno‐associated virus (AAV) vector. His‐tagged survivin was cloned and packaged into the AAV‐rh10 vector.
Correction: Gut dysbiosis is associated with metabolism and systemic inflammation in patients with ischemic stroke
There are values missing from the detection rate column in Table 2 . Please see the corrected Table 2 here. Citation: Yamashiro K, Tanaka R, Urabe T, Ueno Y, Yamashiro Y, Nomoto K, et al. (2017) Correction: Gut dysbiosis is associated with metabolism and systemic inflammation in patients with ischemic stroke. PLoS ONE 12(4): e0176062. https://doi.org/10.1371/journal.pone.0176062 Copyright: © 2017 Yamashiro et al.
Exendin-4 Inhibits Matrix Metalloproteinase-9 Activation and Reduces Infarct Growth After Focal Cerebral Ischemia in Hyperglycemic Mice
Background and Purpose—Admission hyperglycemia is an independent risk factor for poor outcome of ischemic stroke. Amelioration of hyperglycemia by insulin has not been shown to improve the poststroke outcome. Glucagon-like peptide 1 receptor agonists, which modulate glucose levels by stimulating insulin secretion, have been shown to exert cytoprotective effects by inhibiting inflammation and oxidative stress.
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