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c-Jun, c-Jun N-terminal kinase(JNK) and endothelin B (ET B ) receptor have been shown to contribute to the pathogenesis of glaucoma. Previously, we reported that an increase of c-Jun and CCAAT/enhancer binding protein ? (C/EBP?) immunohistostaining is associated with upregulation of the ET B receptor within the ganglion cell layer of rats with elevated intraocular pressure (IOP).
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