Is this you? As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
Claim your profile
Abstract The epigenetic mechanisms by which nutrient overload reprograms lymphatic endothelial cells (LECs) remain unclear. Here we show that metabolic stress drives LECs into a glycolytic state, producing lactate that fuels EP300-catalyzed histone H3K18 lactylation at the VEGFR-3 promoter, thereby enhancing chromatin accessibility and VEGFR-3 transcription.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
