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Articles by Ulf Panzer
Spatiotemporal interaction of immune and renal cells controls glomerular crescent formation in autoimmune kidney disease - Nature Immunology
Abstract Rapidly progressive glomerulonephritis (RPGN) is the most aggressive group of autoimmune kidney diseases and is characterized by glomerular crescent formation with proliferation of parietal epithelial cells (PECs). However, the underlying mechanisms of glomerular crescent formation are incompletely understood.
Type I interferon drives T cell cytotoxicity by upregulation of interferon regulatory factor 7 in autoimmune kidney diseases in mice - Nature Communications
Abstract In anti-neutrophil cytoplasmic antibody-associated vasculitis (AAV) and systemic lupus erythematosus (SLE), glomerulonephritis is a severe kidney complication driven by immune cells, including T cells. However, the mechanisms underlying T cell activation in these contexts remain elusive.
γδ T cells in immune‐mediated kidney disease
Introduction Immune-mediated kidney diseases (glomerulonephritis [GN]) encompass a heterogeneous group of disorders that cause inflammation within the glomerulus and other compartments of the kidney [1-3]. Despite recent important discoveries in this field, the immunopathogenesis of the various forms of immune-mediated glomerular diseases remains to be fully elucidated.
Mucosal-associated invariant T cells contribute to suppression of inflammatory myeloid cells in immune-mediated kidney disease - Nature Communications
Abstract Mucosal-associated invariant T (MAIT) cells have been implicated in various inflammatory diseases of barrier organs, but so far, their role in kidney disease is unclear. Here we report that MAIT cells that recognize their prototypical ligand, the vitamin B2 intermediate 5-OP-RU presented by MR1, reside in human and mouse kidneys.
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