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Publisher Correction: CFTR mediates Cl - transport in osteocytes to sustain cell viability and skeletal homeostasis
Correction to: Nature Communications https://doi.org/10.1038/s41467-026-72349-0, published online 28 April 2026 In Fig. 2e of this article, the Exon 4_KO sequence was inadvertently omitted by the Publisher; the original and corrected figure panel is shown below. The original article has been updated. Incorrected Fig. 2e Corrected Fig.
Plant-derived extracellular vesicle-like nanoparticles: an emerging immunomodulatory strategy for intervertebral disc degeneration
Abstract Intervertebral disc degeneration (IDD), a leading driver of chronic low back pain, has become a major socioeconomic and public health burden due to its high disability rate and substantial treatment costs. Accumulating evidence has redefined IDD as an immune-mediated disorder characterized by the breakdown of immune privilege, infiltration of immune cells, and chronic sterile inflammation driven by innate immune pathways including NF-κB and the NLRP3 inflammasome.
CFTR mediates Cl - transport in osteocytes to sustain cell viability and skeletal homeostasis
Abstract Osteocytes are long-lived with underlying mechanisms largely unknown. Here, we report that osteocyte-specific knockout of cystic fibrosis transmembrane conductance regulator (CFTR) results in excessive osteocyte death, proinflammatory cytokine surge, osteoclast overactivation and bone formation impairment leading to bone loss in adult mice. Consistently in MLO‑Y4 osteocyte‑line, CFTR-knockout causes progressive cell death, which is reversed by CFTR overexpression or medium replenishment.
Defective Cystic Fibrosis Transmembrane Conductance Regulator Accelerates Skeletal Muscle Aging by Impairing Autophagy/Myogenesis
1 Introduction Skeletal muscles can be regenerated through the activation of adult myogenic progenitors for their differentiation into myoblasts and subsequent maturation into multinuclear muscle fibres [1]. Such adult myogenesis process is initiated upon physical injury or other stress/damage to skeletal muscles and complexed with sequential activation of myogenic genes as well as cellular/subcellular organisational changes [2].
Adsorption Kinetics and Mechanism of Pb(II) and Cd(II) Adsorption in Water through Oxidized Multiwalled Carbon Nanotubes
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Enhanced antibacterial and adhesive properties of chitosan/gelatin hydrogel containing berberine hydrochloride and prepolymerized gallic acid exhibiting aggregation‐induced emission
Filename Description pen26398-sup-0001-supinfo.docWord document, 2.3 MB DATA S1. Supporting Information. Please note: The publisher is not responsible for the content or functionality of any supporting information supplied by the authors. Any queries (other than missing content) should be directed to the corresponding author for the article.
Synergistic intrafibrillar/extrafibrillar mineralization of collagen fibrils and scaffolds enhanced by introducing polyacrylamide to PILP for osteogenic differentiation
Abstract Mineralization can help improve the mechanical properties and degradation rates of collagen scaffolds while ensuring good biocompatibility, thereby providing a suitable microenvironment for osteogenic differentiation. Intrafibrillar/extrafibrillar mineralization of collagen is promoted by polymer-induced liquid precursors (PILP). In this study, polyacrylamide (PAM) was introduced to PILP which synergistically mineralized collagen scaffold intrafibrillarly and extrafibrillarly.
Synergistic intrafibrillar/extrafibrillar mineralization of collagen fibrils and scaffolds enhanced by introducing polyacrylamide to PILP for osteogenic differentiation
Abstract Mineralization can help improve the mechanical properties and degradation rates of collagen scaffolds while ensuring good biocompatibility, thereby providing a suitable microenvironment for osteogenic differentiation. Intrafibrillar/extrafibrillar mineralization of collagen is promoted by polymer-induced liquid precursors (PILP). In this study, polyacrylamide (PAM) was introduced to PILP which synergistically mineralized collagen scaffold intrafibrillarly and extrafibrillarly.
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