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Keywords T-LGLL KLRG1 PI3K CITE-seq TCRαβ-seq Introduction T cell large granular lymphocytic leukemia (T-LGLL) is a chronic lymphoproliferative disorder characterized by the clonal expansion of mature terminal effector (TE) CD8+ T large granular lymphocytes (LGLs), leading to hematological complications like neutropenia and anemia.1,2 T cell clones are the hallmark of mature T cell leukemia/lymphoma.
Abstract RNA-binding proteins (RBPs) are critical regulators for RNA transcription and translation. As a key member of RBPs, ELAV-like family protein 2 (CELF2) has been shown to regulate RNA splicing and embryonic hematopoietic development and was frequently seen dysregulated in acute myeloid leukemia (AML). However, the functional role(s) of CELF2 in hematopoiesis and leukemogenesis has not been fully elucidated.
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