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Articles by Yangjun Yang
TMEM192 is a calcineurin-regulated membrane receptor for ubiquitin-independent lysophagy
Abstract Lysophagy is a selective form of autophagy responsible for clearing damaged lysosomes. Although existing models predominantly highlight ubiquitin-dependent mechanisms, here we identify the lysosomal membrane protein TMEM192 as a key ubiquitin-independent receptor for lysophagy. We demonstrate that lysophagy persists in cells even when protein ubiquitination is inhibited or autophagy adaptors are deleted.
Mitochondria-Associated Membranes as Key Regulators in Cellular Homeostasis and the Potential Impact of Exercise on Insulin Resistance
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Research Progress on Lipophagy-Mediated Exercise Intervention in Non-Alcoholic Fatty Liver Disease
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
IJMS | Free Full-Text | High-Intensity Interval Training Ameliorates High-Fat Diet-Induced Metabolic Disorders via the Cyclic GMP-AMP Synthase-Stimulator of Interferon Gene Signaling Pathway
1. Introduction The incidence of metabolic diseases such as obesity, type 2 diabetes mellitus (T2DM), and cardiovascular diseases is increasing year by year due to population aging and poor lifestyle. Therefore, it is urgent to study the pathological mechanisms and prevention strategies of metabolic diseases. Notably, in recent years, research on metabolic diseases has increasingly focused on the field of immune inflammation.
FUNDC1: An Emerging Mitochondrial and MAMs Protein for Mitochondrial Quality Control in Heart Diseases
Abstract : Heart diseases (HDs) are the leading cause of mortality worldwide, with mitochondrial dysfunction being a significant factor in their development. The recently discovered mitophagy receptor, FUNDC1, plays a critical role in regulating the homeostasis of the Mitochondrial Quality Control (MQC) system and contributing to HDs. The phosphorylation of specific regions of FUNDC1 and varying levels of its expression have been shown to have diverse effects on cardiac injury.
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