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Abstract LAT3 (SLC43A1) mediates sodium-independent transport of neutral and branched-chain amino acids and participates in both physiological and pathological processes, including development and cancer. Dysregulation of LAT3 function is associated with disease, yet the regulatory mechanism of LAT3 remains poorly defined.
Muhammad Idrees Afridi Research Center for Mathematical Modeling and Simulation, Hanjiang Normal University, Shiyan, China Department of Mathematics, Saveetha School of Engineering, SIMATS, Saveetha University, Chennai, Tamil Nadu, India Applied Science Research Center, Applied Science Private University, Amman, Jordan Search for more papers by this author
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