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Articles by Yuji Ueno
Microbial lipopolysaccharide‐induced inflammation contributes to cognitive impairment and white matter lesion progression in diet‐induced obese mice with chronic cerebral hypoperfusion
Abstract Aims White matter lesions (WMLs) are involved in the pathological processes leading to cognitive decline and dementia. We examined the mechanisms underlying the exacerbation of ischemia-induced cognitive impairment and WMLs by diet-induced obesity, including lipopolysaccharide (LPS)-triggered neuroinflammation via toll-like receptor (TLR) 4.
Antifungal susceptibility of dermatophytes from racehorses in Japan
Brief Communication Ryousuke Watanabe, Department of Veterinary Dermatology, Nihon University College of Bioresource Sciences, Fujisawa, Kanagawa, Japan Contribution: Conceptualization, Data curation, Formal analysis, Investigation Search for more papers by this author Hikaru Huruta, Department of Veterinary Dermatology, Nihon University College of Bioresource Sciences, Fujisawa, Kanagawa, Japan Contribution: Conceptualization, Formal analysis Search for more papers by this author Yuji Ueno,...
Correction: Gut dysbiosis is associated with metabolism and systemic inflammation in patients with ischemic stroke
There are values missing from the detection rate column in Table 2 . Please see the corrected Table 2 here. Citation: Yamashiro K, Tanaka R, Urabe T, Ueno Y, Yamashiro Y, Nomoto K, et al. (2017) Correction: Gut dysbiosis is associated with metabolism and systemic inflammation in patients with ischemic stroke. PLoS ONE 12(4): e0176062. https://doi.org/10.1371/journal.pone.0176062 Copyright: © 2017 Yamashiro et al.
Emerging Risk Factors for Recurrent Vascular Events in Patients With Embolic Stroke of Undetermined Source
Original Contribution Emerging Risk Factors for Recurrent Vascular Events in Patients With Embolic Stroke of Undetermined Source Yuji Ueno, Kazuo Yamashiro, Ryota Tanaka, Takuma Kuroki, Kenichiro Hira, Naohide Kurita, Takao Urabe and Nobutaka Hattori
Exendin-4 Inhibits Matrix Metalloproteinase-9 Activation and Reduces Infarct Growth After Focal Cerebral Ischemia in Hyperglycemic Mice
Background and Purpose—Admission hyperglycemia is an independent risk factor for poor outcome of ischemic stroke. Amelioration of hyperglycemia by insulin has not been shown to improve the poststroke outcome. Glucagon-like peptide 1 receptor agonists, which modulate glucose levels by stimulating insulin secretion, have been shown to exert cytoprotective effects by inhibiting inflammation and oxidative stress.
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