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Antimicrobial resistance (AMR) remains one of the most urgent public health threats of our time. By rendering existing antibiotics less effective or even useless, AMR threatens to roll back decades of medical progress, increasing patient mortality and morbidity, prolonging hospital stays, and inflating healthcare costs.
Disclaimer: Early release articles are not considered as final versions. Any changes will be reflected in the online version in the month the article is officially released. Aurélie Hennebique1 , Yvan Caspar1, Max Maurin, Sandrine Boisset, Isabelle Pelloux, Maria Pilar Gallego-Hernanz, Christophe Burucoa, France Cazenave-Roblot, Chloé Plouzeau, and Blandine Rammaert Author affiliations: Université Grenoble Alpes, Grenoble (A. Hennebique, Y. Caspar, M. Maurin, S.
Pseudomonas aeruginosa is responsible for chronic respiratory tract colonisation and acute exacerbations in cystic fibrosis (CF) patients. This Gram-negative bacterium often develops multidrug resistance, which represents a therapeutic challenge. Our objective was to characterize the phenotypic and genetic β-lactam resistance traits of P. aeruginosa strains isolated from CF patients followed at Grenoble Alpes university hospital.The susceptibility to β-lactam compounds of 123 P.
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