1 Introduction Sarin and soman, possessing reactive phosphorus-fluorine bonds, are highly lethal and fast-acting nerve agents. These agents can irreversibly inhibit the activity of acetylcholinesterase (AChE), triggering excessive accumulation of the neurotransmitter acetylcholine, ultimately resulting in respiratory failure and human death. Given their extreme toxicity and rapid pathogenicity, nerve agents pose a severe and persistent threat to public health and global safety [1, 2].
