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Abstract Vascular cognitive impairment and dementia (VCID) is characterized by extensive subcortical white matter injury and progressive sensorimotor and cognitive impairment. Post-translational modification of UCHL1 at cysteine 152 (C152) impairs repair of white matter injury and recovery of function after ischemia. We hypothesized that mutation of UCHL1 C152 would ameliorate white matter injury and cognitive impairment in VCID.
As a journalist, you can create a free Muck Rack account to customize your profile, list your contact preferences, and upload a portfolio of your best work.
