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Abstract This brief research report describes eight children with RESLES treated at a single tertiary hospital. Etiologies included influenza A infection, respiratory syncytial virus with adenovirus coinfection, clinically suspected enterovirus-related herpangina, autoimmune glial fibrillary acidic protein astrocytopathy (GFAP-A), and one severe infantile encephalopathy with RESLES-like callosal involvement.
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