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Articles by Adonis Sfera
Cytokines and Madness: A Unifying Hypothesis of Schizophrenia Involving Interleukin-22
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Cytokines and Madness: A Unifying Hypothesis of Schizophrenia Involving Interleukin-22
Preprint Review Version 1 This version is not peer-reviewed Version 1 : Received: 1 November 2024 / Approved: 4 November 2024 / Online: 5 November 2024 (09:14:56 CET) Sfera, A.; Thomas, K. A.; Anton, J. Cytokines and Madness: A Unifying Hypothesis of Schizophrenia Involving Interleukin-22. Preprints 2024, 2024110219. https://doi.org/10.20944/preprints202411.0219.v1 Sfera, A.; Thomas, K. A.; Anton, J. Cytokines and Madness: A Unifying Hypothesis of Schizophrenia Involving Interleukin-22.
Novel Insights into Psychosis and Antipsychotic Interventions: From Managing Symptoms to Improving Outcomes
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
The Future of Antipsychotic Interventions: From Managing Symptoms to Improving Outcomes
Preprint Review Version 1 This version is not peer-reviewed Version 1 : Received: 26 March 2024 / Approved: 27 March 2024 / Online: 28 March 2024 (12:24:09 CET) Sfera, A.; Imran, H.; Sfera, D.O.; Anton, J.J.; Kozlakidis, Z.; Hazan, S. The Future of Antipsychotic Interventions: From Managing Symptoms to Improving Outcomes. Preprints 2024, 2024031692. https://doi.org/10.20944/preprints202403.1692.v1 Sfera, A.; Imran, H.; Sfera, D.O.; Anton, J.J.; Kozlakidis, Z.; Hazan, S.
Insomnia in Forensic Detainees: Is Salience Network the Common Pathway for Sleep, Neuropsychiatric, and Neurodegenerative Disorders?
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Sleepless in Captivity: Insomnia Kills the Von Economo Neurons
Preprint Review Version 1 This version is not peer-reviewed Version 1 : Received: 27 February 2024 / Approved: 27 February 2024 / Online: 28 February 2024 (04:50:10 CET) Sfera, A.; Thomas, K.A.; Ogunjale, I.A.; Jafri, N.; Bota, P.G. Sleepless in Captivity: Insomnia Kills the Von Economo Neurons. Preprints 2024, 2024021560. https://doi.org/10.20944/preprints202402.1560.v1 Sfera, A.; Thomas, K.A.; Ogunjale, I.A.; Jafri, N.; Bota, P.G. Sleepless in Captivity: Insomnia Kills the Von Economo Neurons.
Of Soldiers and Their Ghosts: Are We Ready for a Review of PTSD Evidence?
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
Receptor-Independent Therapies for Forensic Detainees with Schizophrenia-Dementia Comorbidity
1. Introduction The population worldwide is aging at a rapid pace and so are forensic detainees with severe psychiatric illnesses, including schizophrenia (SCZ) and schizophrenia-like disorders (SLDs). In forensic institutions, there are two subpopulations of elderly patients with neurocognitive disorders: career criminals with lifelong mental illness and elderly first offenders without previous psychiatric history.
Long COVID as a Tauopathy: Of “Brain Fog” and “Fusogen Storms”
1. Introduction The COVID-19 pandemic may be succeeded by prolonged sequelae that could affect the life quality of many patients while at the same time increasing medical complications and healthcare expenditures. Cognitive impairment, one of the lingering symptoms, often referred to by patients as “brain fog”, remains poorly defined; however, viral presence in reservoirs and the “seeding” of pathological Tau may account for cognitive difficulties.
Six Decades of Dopamine Hypothesis: Is Aryl Hydrocarbon Receptor the New D2?
Abstract : In 1957, Arvid Carlsson discovered that dopamine, at the time believed to be nothing more than a norepinephrine precursor, was a brain neurotransmitter in and of itself. By 1963, postsynaptic dopamine blockade had become the cornerstone of psychiatric treatment as it appeared to have deciphered the “chlorpromazine enigma”, a 1950s term, denoting the action mechanism of antipsychotic drugs.
The Role of Lactylation in Mental Illness: Emphasis on Microglia
1. Introduction Severe neuropsychiatric illnesses, including schizophrenia and affective disorders, have been associated with brain metabolic disturbances, including excessive aerobic glycolysis and upregulated lactate, emphasizing that in the central nervous system (CNS) bioenergetics and information processing are highly intertwined [1,2,3,4].
Frontiers | Using substantial reductant concentration with chelation therapy to enhance small aggregate dispersal, iron mobilization, and its clearance in neurodegenerative diseases
Introduction Elevated iron levels are associated with several neurodegenerative diseases through numerous in vivo and in vitro studies (Singh et al., 2014). Excess iron deposition has been mapped to specific regions in the brain associated with particular neurodegenerative disease characteristics through histology and magnetic resonance imaging. Cultured cells behave abnormally when exposed to excess iron as do organelles.
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