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Ainara Meler on Muck Rack

Ainara Meler

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HDAC7 is a major contributor in the pathogenesis of infant t(4;11) proB acute lymphoblastic leukemia

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By Juan L. Trincado, Ronald W. Stam, Alba Azagra, Ainara Meler
| Nature Verified
Fig. 1: HDAC7 exerts a leukemia suppressive phenotype in t(4;11) proB ALL cells, and its absence associates with poor prognosis in proB ALL infants. Fig. 2: Enforced overexpression of HDAC7 partially reverts the expression pattern of genes affected by t(4;11) translocation in proB ALL. References 1. Pui CH, Mullighan CG, Evans WE, Relling MV. Pediatric acute lymphoblastic leukemia: where are we going and how do we get there? Blood. 2012;120:1165–74. CAS Article Google Scholar 2.
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