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Akbar L. Marzan

As seen in: Wiley Online Library, MDPI
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Articles

Unravelling the Role of Cancer Cell-Derived Extracellular Vesicles in Muscle Atrophy, Lipolysis, and Cancer-Associated Cachexia

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By Akbar L. Marzan, Sai V Chitti
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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Proteomics analysis of C2C12 myotubes treated with atrophy inducing cancer cell‐derived factors

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By Akbar L. Marzan, Sai V Chitti, Sriram Gummadi, Taeyoung Kang
| Wiley Online Library Verified
Ampd3 AMP deaminase 3 AMP Adenosine monophosphate CM Conditioned media Cox5b Cytochrome c oxidase subunit 5B ECM Extracellular matrix EVs Extracellular vesicles FDR False discovery rate FOXO Forkhead box HD Heat deactivated IFN-γ Interferon-gamma IL-8 Interleukin-8 IMP Inosine monophosphate LPS Lipopolysaccharide MyHC Myosine heavy chain NO Nitric oxide PDGFRβ Platelet-derived growth factor receptor-beta ROS Reactive oxygen species Sdf2l1 Stromal cell-derived factor 2-like 1 TGF-β...
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Proteomic analysis of the small extracellular vesicles and soluble secretory proteins from cachexia inducing and non‐inducing cancer cells

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By Sai V Chitti, Pamali Fonseka, Taeyoung Kang, Akbar L. Marzan
| Wiley Online Library Verified
Proteins implicated in cancer cachexia are enriched in the secretome of C26 cells To identify the biological processes that are enriched among proteins identified from C26 CM, proteins that were statistically significant with fold change greater than ± 2 in the secretome of C26 and EL4 cells were subjected to functional enrichment analysis using FunRich [25, 26].
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