Alex Noble on Muck Rack

Alex Noble

As seen in: The Hollywood Reporter, MSN Canada, Nature, Yahoo Canada, Yahoo News Malaysia, Yahoo Singapore, Newsweek, The Wrap, Yahoo, Royal Society of Chemistry, BioRxiv and
movie person • usc & sdsu alumna
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Articles

Myosin forces remodel F-actin for mechanosensitive protein recognition

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By Ayala G Carl, Matthew Reynolds, Xiaoyu Sun, Pinar Gurel, Keith Hamilton, Lin Mei, John W. Watters, Yasuharu Takagi, Alex Noble, James R. Sellers, Gregory M. Alushin, Donovan Y. Z. Phua
| Nature Verified
Abstract Cells interface mechanically with their surroundings through cytoskeleton-linked adhesions1,2, which enable them to sense physical cues that instruct development and drive diseases such as cancer3,4,5. Contractile forces generated by myosin motor proteins6,7 mediate these mechanical signal transduction processes through unknown protein structural mechanisms.
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Influence of lung extracellular matrix from non-IPF and IPF donors on primary human lung fibroblast biology

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By Mohammadhossein Dabaghi, Ryan Singer, Alex Noble, David Gonzalez-Martinez
| HAL
Article Dans Une Revue Biomaterials Science Année : 2025 Influence of lung extracellular matrix from non-IPF and IPF donors on primary human lung fibroblast biology Domaines hal-05053846 , version 1 (02-05-2025) HAL Id : hal-05053846 , version 1 DOI : 10.1039/d4bm00906a PUBMED : 39968884 WOS : 001424857300001 Mohammadhossein Dabaghi, Ryan Singer, Alex Noble, Aidee Veronica Arizpe Tafoya, David A González-Martínez, et al..
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Influence of Lung Extracellular Matrix from non-IPF and IPF Donors on Primary Human Lung Fibroblast Biology

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By Mohammadhossein Dabaghi, Ryan Singer, Alex Noble, Tamaghna Gupta
| Royal Society of Chemistry Verified
Influence of Lung Extracellular Matrix from non-IPF and IPF Donors on Primary Human Lung Fibroblast Biology Fibrosis, a pathological hallmark of various chronic diseases, involves the excessive accumulation of extracellular matrix (ECM) components leading to tissue scarring and functional impairment. Understanding how cells interact with the ECM in fibrotic diseases such as idiopathic pulmonary fibrosis (IPF), is crucial for developing effective therapeutic strategies.
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