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Abstract γδ T cells are important for host defense at the respiratory mucosa, acting directly or through interactions with other cells. However, how γδ T cells influence other immune cells in the lung remains unclear. Using a genetically engineered mouse model of lung cancer, we show that tumors drive expansion of both CD27+ and CD27− γδ T cells.
Abstract Protecting mucosal barriers, γδ T cells hold promise for the development of new cancer immunotherapies. In mice, γδ T cells can largely be segregated into CD27+ and CD27– cells, and their functions are modulated by interactions with surrounding cells. However, which cells communicate directly with γδ T cells in lung adenocarcinoma remains unknown.
Article in Press Enteric helminth-induced type I interferon signaling protects against pulmonary virus infection through interaction with the microbiota, , , Paul M. FitchxPaul M. FitchSearch for articles by this authorAffiliationsMRC–Centre for Inflammation Research, University of Edinburgh, Edinburgh, United KingdomChild Life and Health, University of Edinburgh, Edinburgh, United Kingdom, PhD, , , , , , Michael R. EdwardsxMichael R.
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