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Abstract Although Alzheimer’s disease (AD) has a polygenic architecture, with numerous associated single-nucleotide polymorphisms (SNPs) identified, the contribution of structural variants (SVs) is unclear.
To use the Enhanced view of this article, please enable JavaScript on in your browser and refresh the page. PreviewHomeostatic Scaling of AMPA Receptors by Semaphorin Amy K.Y. FuxAmy K.Y. FuSearch for articles by this authorAffiliationsDivision of Life Science, State Key Laboratory of Molecular Neuroscience and Molecular Neuroscience Center, The Hong Kong University of Science and Technology, Clear Water Bay, Kowloon, Hong Kong, China, Nancy Y. IpxNancy Y.
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