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Anbarasu Lourdusamy on Muck Rack

Anbarasu Lourdusamy

As seen in: Color Research & Application, Nature
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Articles

Hippocampal Cofilin and CFL1 gene variants are linked to Alcohol Use Disorder phenotypes - Molecular Psychiatry

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By Ahmad Salamian, Roberto Pagano, Monika Puchalska, Bartosz Wojtas, Anna Cały, Christiane Mühle, Bernd Lenz, Johannes Kornhuber, Anbarasu Lourdusamy, Christian Müller, Małgorzata Grochowicz
| Nature Verified
Abstract Alcohol use disorder (AUD) is characterized by pathological motivation to consume alcohol and cognitive inflexibility, leading to excessive alcohol seeking and use. In this study, we investigated the molecular correlates of impaired extinction of alcohol seeking during forced abstinence using a mouse model of AUD in the automated IntelliCage social system. This model distinguished AUD-prone and AUD-resistant animals based on the presence of ≥2 or <2 criteria of AUD, respectively.
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Acid sphingomyelinase activity suggests a new antipsychotic pharmaco-treatment strategy for schizophrenia - Molecular Psychiatry

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By Christiane Mühle, Fabian Schumacher, Stephan von Hörsten, Arif B. Ekici, Johannes Kornhuber, Jae-Sung Bae, Anbarasu Lourdusamy
| Nature Verified
Abstract Schizophrenia is a chronic and severe mental disorder. It is currently treated with antipsychotic drugs (APD). However, APD’s work only in a limited number of patients and may have cognition impairing side effects. A growing body of evidence points out the potential involvement of abnormal sphingolipid metabolism in the pathophysiology of schizophrenia.
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3D hydrogels reveal medulloblastoma subgroup differences and identify extracellular matrix subtypes that predict patient outcome

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By Franziska Linke, Macha Aldighieri, Anbarasu Lourdusamy
| Color Research & Application Verified
Abstract Medulloblastoma (MB) is the most common malignant brain tumour in children and is subdivided into four subgroups: WNT, SHH, Group 3, and Group 4. These molecular subgroups differ in their metastasis patterns and related prognosis rates. Conventional 2D cell culture methods fail to recapitulate these clinical differences. Realistic 3D models of the cerebellum are therefore necessary to investigate subgroup‐specific functional differences and their role in metastasis and chemoresistance.
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