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Annaick Carles

As seen in: Future Medicine
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Articles

The Pathognomonic FOXL2 C134W Mutation Alters DNA-Binding Specificity

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By Annaick Carles, Genny Trigo-Gonzalez, Qi Cao, S.-W. Grace Cheng
| cancerres.aacrjournals.org
Genome and Epigenome Annaïck Carles, Genny Trigo-Gonzalez, Qi Cao, S.-W. Grace Cheng, Michelle Moksa, Misha Bilenky, David G. Huntsman, Gregg B. Morin and Martin Hirst DOI: 10.1158/0008-5472.CAN-20-0104 Published September 2020 This article requires a subscription to view the full text. You may purchase access to this article or login to access your subscription using the links below.
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Epigenomic programming in early fetal brain development

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By Luolan Li, Cecile L. Maire, Misha Bilenky, Annaick Carles
| Future Medicine Verified
Transcription is the driver of cell differentiation and specification involving multilateral interactions between epigenetic modifications and transcription factors. While significant progress has been made in defining transcriptional programs within distinct brain cell types [1–5], we still lack comprehensive knowledge of how these programs are shaped by mitotically inherited epigenetic states during brain development.
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The pathognomonic FOXL2 C134W mutation alters DNA binding specificity

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By Annaick Carles, Genny Trigo-Gonzalez, Rachelle Cao, S.-W. Grace Cheng
| cancerres.aacrjournals.org
Research Article Annaïck Carles, Genny Trigo-Gonzalez, Rachelle Cao, S.-W. Grace Cheng, Michelle Moksa, Misha Bilenky, David G. Huntsman, Gregg B Morin and Martin Hirst DOI: 10.1158/0008-5472.CAN-20-0104 Abstract The somatic missense point mutation c.402C>G (p.C134W) in the FOXL2 transcription factor is pathognomonic for adult-type granulosa cell tumors (AGCT) and a diagnostic marker for this tumor type.
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