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Anwarul Ferdous on Muck Rack

Anwarul Ferdous

As seen in: Nature
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Articles

VCAM-1/Ezrin axis antagonizes myocardial damage in ischemia-reperfusion injury - Cell Death & Differentiation

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By Anwarul Ferdous, Ariel Diaz, Daniel Daou, Nan Jiang, Herman I. May, Jan-Bernd Funcke, Magda C Díaz-Vesga, Lorena Garcia Verified, Lorena García, Mario Chiong, Dian J. Cao, Thomas Gillette, Sergio Lavandero, Diana Dad Zada, Juan A. Daniel-Olivas, Jafet Ortiz-Quintero
| Nature Verified
Abstract Vascular cell adhesion molecule 1 (VCAM-1), a known downstream target of the Forkhead box O (FoxO) family of transcription factors, has well-established roles in development, cell-cell interactions, and cell survival. However, the specific role and mechanisms whereby VCAM-1 governs cardiomyocyte homeostasis in ischemic heart disease are incompletely understood.
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Xbp1s-FoxO1 axis governs lipid accumulation and contractile performance in heart failure with preserved ejection fraction

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By Gabriele G. Schiattarella, Francisco Altamirano, Anwarul Ferdous, Hande Piristine
| Nature Verified
Abstract Heart failure with preserved ejection fraction (HFpEF) is now the dominant form of heart failure and one for which no efficacious therapies exist. Obesity and lipid mishandling greatly contribute to HFpEF. However, molecular mechanism(s) governing metabolic alterations and perturbations in lipid homeostasis in HFpEF are largely unknown.
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FoxO1-Dio2 signaling axis governs cardiomyocyte thyroid hormone metabolism and hypertrophic growth

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By Zhao Wang, Xiang Luo, Gabriele G. Schiattarella, Anwarul Ferdous
| Nature Verified
Abstract Forkhead box O (FoxO) proteins and thyroid hormone (TH) have well established roles in cardiovascular morphogenesis and remodeling. However, specific role(s) of individual FoxO family members in stress-induced growth and remodeling of cardiomyocytes remains unknown. Here, we report that FoxO1, but not FoxO3, activity is essential for reciprocal regulation of types II and III iodothyronine deiodinases (Dio2 and Dio3, respectively), key enzymes involved in intracellular TH metabolism.
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