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Ashlyn Bartlett Conant on Muck Rack

Ashlyn Bartlett Conant

As seen in: Wiley Online Library, MDPI, BioRxiv
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Articles

Interferon beta drives therapy resistance in a patient-derived model of high-grade serous ovarian cancer

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By Ashlyn Bartlett Conant, Tise Suzuki, Kiera McGivney
| Wiley Online Library Verified
CR resistant HGSOC high-grade serous ovarian cancer IFN interferon IFN-1 interferon type 1 IFNα interferon Alpha IFNβ interferon Beta IRDS interferon-related DNA damage resistance signature ISG interferon-stimulated gene ISGF3 interferon-stimulated gene factor 3 PDX patient-derived xenograft SE sensitive TCGA The Cancer Genome Atlas u-ISGF3 unphosphorylated Interferon-stimulated gene factor 3 1 Introduction High-grade serous ovarian carcinoma (HGSOC) remains one of the deadliest gynecologic...
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A Stem-like Patient-Derived Ovarian Cancer Model of Platinum Resistance Reveals Dissociation of Stemness and Resistance

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By Tise Suzuki, Ashlyn Bartlett Conant, Yeonkyu Jung, Ryan Bax
| MDPI
All articles published by MDPI are made immediately available worldwide under an open access license. No special permission is required to reuse all or part of the article published by MDPI, including figures and tables. For articles published under an open access Creative Common CC BY license, any part of the article may be reused without permission provided that the original article is clearly cited. For more information, please refer to https://www.mdpi.com/openaccess.
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Modeling platinum resistance in a stem-like patient-derived ovarian cancer sample

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By Tise Suzuki, Ryan Bax, Ashlyn Bartlett Conant, Yeonkyu Jung
| BioRxiv
Abstract Background Chemoresistance and tumor recurrence remain a significant challenge in ovarian cancer. Particularly in the context of platinum resistance, many mechanisms have been identified, including the activation of cellular processes like epithelial-mesenchymal transition (EMT), which generates cells with stemness characteristics. Current models of platinum resistance are limited or not adequate representations of the heterogeneity of the disease.
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