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Asim K Duttaroy on Muck Rack

Asim K Duttaroy

Oslo
As seen in: Nature, Frontiers, Frontiers in Immunology, MDPI, BioRxiv
Professor, Faculty of Medicine, University of Oslo, Clinical Research and teaching, Editor of several journals, Advisor to several pharma asimduttaroy.com
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Articles

An updated review on the role of extracellular vesicles in immune system modulation in breast cancer with special emphasis on immune checkpoint regulators

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By Surajit Pathak, Arunkumar Radhakrishnan, Antara Banerjee, Asim K Duttaroy, Jayenth Jayachandran, Zulya Maizetova
| Frontiers in Immunology
Abstract Breast cancer progression and resistance to therapy are strongly influenced by immune evasion within the tumor microenvironment. Immune checkpoint signaling is a major mechanism by which cancer cells evade immune surveillance, thereby promoting tumor progression and reducing the effectiveness of immunotherapy.
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Leptin-specific epigenetic modulation of preterm cord blood serves as a candidate biomarker for obesity

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By Navya Sree Boga, Amit Banerjee Verified, Saikanth Varma, Archana Molangiri, Syeda Farhana, Santosh Kumar Banjara, Nitasha Bagga, Asim K Duttaroy, Sanjay Basak Verified
| Nature Verified
Abstract Leptin communicates the body’s energy utilisation and maintains a stable body weight. Preterm neonates often show rapid compensatory catch-up growth, which may increase the risk of obesity later in life. Although leptin regulates adiposity, its epigenetic role in the obesity risk of preterm neonates remains unclear. Global and LEP promoter methylation, leptin and IGF1 levels, and expression of LEP and IL6 genes were measured in umbilical cord blood from preterm and term deliveries.
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Transgenerational exposure to plastics-derived endocrine‐disrupting bisphenol A and its analogs on male infertility: impact of gut dysbiosis and epigenetic regulation

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By Sanjay Basak Verified, Saikanth Varma, Asim K Duttaroy, Subhalakshmi Nag
| Frontiers
Abstract Global declines in male fertility, characterized by reduced sperm count, motility, and quality, raised concerns about environmental exposures to estrogen-mimicking endocrine-disrupting chemicals (EDCs), including bisphenol A (BPA), and reproductive dysfunction. BPA exposure in vivo has been shown to alter gut microbial composition, diversity, and metabolites, leading to dysbiosis.
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