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Aurélia Barascu on Muck Rack

Aurélia Barascu

As seen in: Nature, MDPI, BioRxiv
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Articles

Hog1 acts in a Mec1-independent manner to counteract oxidative stress following telomerase inactivation in Saccharomyces cerevisiae - Communications Biology

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By María Teixeira, Aurélia Barascu
| Nature Verified
Abstract Replicative senescence is triggered when telomeres reach critically short length and activate permanent DNA damage checkpoint-dependent cell cycle arrest. Mitochondrial dysfunction and increase in oxidative stress are both features of replicative senescence in mammalian cells. However, how reactive oxygen species levels are controlled during senescence is elusive.
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Hog1 acts in a Mec1-independent manner to counteract oxidative stress following telomerase inactivation

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By Aurélia Barascu, Bechara Zeinoun, María Teixeira
| BioRxiv
Abstract Replicative senescence is triggered when telomeres reach critically short length and activate permanent DNA damage checkpoint-dependent cell cycle arrest. Mitochondrial dysfunction and increase in oxidative stress are both features of replicative senescence in mammalian cells.
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TERRA and Telomere Maintenance in the Yeast Saccharomyces cerevisiae

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By María Teixeira, Aurélia Barascu, Maria Teresa, Bechara Zeinoun
| MDPI
This is an early access version, the complete PDF, HTML, and XML versions will be available soon. Open AccessReview by Bechara Zeinoun , Maria Teresa Teixeira and Aurélia Barascu * Laboratoire de Biologie Moléculaire et Cellulaire des Eucaryotes, CNRS, UMR8226, Institut de Biologie Physico-Chimique, Sorbonne Université, F-75005 Paris, France * Author to whom correspondence should be addressed.
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