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Barbara Leggett on Muck Rack

Barbara Leggett

As seen in: Nature, Cambridge University Press & Assessment, Journal of Medical Case Reports, Medical Journal of Australia (MJA)
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Articles

Attenuation of the CpG island methylator phenotype and lack of WNT signalling activation restrains Kras mutant intestinal neoplasia - British Journal of Cancer

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By Lochlan J. Fennell, cheng Liu, Alexandra Kane, Simon Manuel Tria, Jennifer Borowsky, Lu Chai, Sarron Randall-Demllo, Diane M. McKeone, Catherine Bond, Barbara Leggett, Vicki Whitehall
| Nature Verified
Abstract Serrated neoplasia arises from serrated precursor lesions. Hyperplastic polyps commonly activate MAPK signalling, initiated by BRAF or KRAS mutation, but premalignant KRAS-mutant sessile serrated lesions are rare. Here, we model Kras- and Braf-mutant neoplasia in vivo comparing histological, transcriptomic, and epigenetic changes. Temporospatial activation of oncogenic BrafV637 or KrasG12D was induced in murine intestine.
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Dietary pattern and lifestyle risk factors for sessile serrated precursors to colorectal cancer

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By Elizabeth Johnston, Torukiri I Ibiebele, David Hewett, Barbara Leggett
| Cambridge University Press & Assessment
Hostname: page-component-5b777bbd6c-v4w92 Total loading time: 0 Render date: 2025-06-24T13:06:20.392Z Has data issue: false hasContentIssue false Accepted manuscript Published online by Cambridge University Press: 24 June 2025 Elizabeth A. Johnston [Opens in a new window] , Show author details Abstract Keywords bowel cancercancer riskcolorectal polypsepidemiologysessile serrated adenoma Type Research Article Information British Journal of Nutrition , Accepted manuscript , pp.
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Diabetes mellitus and the progression of non‐alcoholic fatty liver disease to decompensated cirrhosis: a retrospective cohort study

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By James O'Beirne, Barbara Leggett, Gunter Hartel, Richard Skoien
| Medical Journal of Australia (MJA) Verified
Med J Aust || doi: 10.5694/mja2.52104 Published online: 9 October 2023 About five million Australians have non‐alcoholic fatty liver disease (NAFLD), and the number is projected to rise to seven million by 2030.1 NAFLD can progress to adverse hepatic disease, such as cirrhosis and its complications, including hepatocellular carcinoma.
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