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Barbara M. Braunger on Muck Rack

Barbara M. Braunger

As seen in: Nature, Science Magazine
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Articles

VEGFR2 deletion increases susceptibility to photoreceptor degeneration through glial-neuronal interaction

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By Christina Bielmeier, Sabrina Schmitt, Andrea E. Dillinger, Herbert Jägle, Christine von Toerne, Stefanie Hauck, Süleyman Ergün, Ernst R. Tamm, Anja Schlecht, Andreas Neueder, Barbara M. Braunger, Verena Lehr, Anita Grundl
| Nature Verified
Abstract Intraocular anti-VEGFA injections are frequently used to counteract neovascularization in diseases such as the neovascular form of age-related macular degeneration (AMD). However, in the clinical context, patients with atrophic (dry) and neovascular (wet) AMD in the same eye will receive anti-VEGFA injections, but there are reports that this severely promotes the development of atrophic areas in dry AMD.
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GPR124 regulates hyaloid blood vessel regression and is associated with endothelial-mesenchymal transition

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By Laura Hannig, Robin Heiden, Süleyman Ergün, Barbara M. Braunger, Mario Vallon
| Nature Verified
Abstract Transition from the transient hyaloid vasculature to the mature retinal vascular network is a critical step in mammalian eye development. While WNT7B signaling in endothelial cells regulates programmed hyaloid vessel regression, the specific receptors and downstream mechanisms remain poorly defined. Here, we identify GPR124, a ligand-specific co-receptor for WNT7A/B, as a key regulator of this process.
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The Blood-ocular Barriers and their Dysfunction: Anatomy, Physiology, Pathology

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By Braunger BM, Barbara M. Braunger, Andreas Gießl, Schlötzer-Schrehardt U
| ncbi.nlm.nih.gov
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