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Modifications in nitric oxide and superoxide anion metabolism induced by fructose overload in the heart are prevented by (-)-epicatechin
Fructose overload promotes functional and metabolic derangements in humans and in animal experimental models. Evidence suggests that dietary flavonoids have the ability to prevent/attenuate the development of metabolic diseases. In this work we investigated the effects of (–)-epicatechin on the modifications induced by fructose overload in the rat heart in terms of nitric oxide and superoxide metabolism.
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