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Barbara Walch-Rückheim on Muck Rack

Barbara Walch-Rückheim

As seen in: Journal of Translational Medicine
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Articles

PD-L1+ CD49f+ CD133+ Circulating tumor cells predict outcome of patients with vulvar or cervical cancer after radio- and chemoradiotherapy - Journal of Translational Medicine

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By Erich Franz, Selina Gies, Patrick Melchior, Istvan Molnar, István Gábor Molnár, Russalina Stroeder, Maike Pohlers, Moritz Schäfer, Laura Theobald, Martina Sester, Barbara Walch-Rückheim, Gregor Olmes, Tanja Tänzer, Erich Franz Solomayer
| Journal of Translational Medicine Verified
Peripheral blood samples were obtained from 40 patients with vulvar cancer and 115 patients with cervical cancer before and during different therapeutic approaches as well as samples from untreated female age-matched immunocompetent controls (HC) and used for monitoring of CTC subsets constitution. A subpopulation of n = 40 age-matched HC were used in experiments for detection of CTCs via immunofluorescence (IF), respectively. In flow cytometry analysis a total of n = 115 HC were analyzed.
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Cervical Cancer-Instructed Stromal Fibroblasts Enhance IL23 Expression in Dendritic Cells to Support Expansion of Th17 Cells

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By Barbara Walch-Rückheim, Russalina Ströder, Laura Theobald, Jennifer Pahne-Zeppenfeld
| cancerres.aacrjournals.org
Tumor Biology and Immunology Barbara Walch-Rückheim, Russalina Ströder, Laura Theobald, Jennifer Pahne-Zeppenfeld, Subramanya Hegde, Yoo-Jin Kim, Rainer Maria Bohle, Ingolf Juhasz-Böss, Erich-Franz Solomayer and Sigrun Smola DOI: 10.1158/0008-5472.CAN-18-1913 Published April 2019 Abstract Persistent infection with high-risk human papillomavirus (HPV) is a prerequisite for the development of cervical cancer.
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Cervical cancer-instructed stromal fibroblasts enhance IL-23 expression in dendritic cells to support expansion of Th17 cells

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By Laura Theobald, Barbara Walch-Rückheim, Russalina Ströder, Jennifer Pahne-Zeppenfeld
| cancerres.aacrjournals.org
Research Article Barbara Walch-Rückheim, Russalina Ströder, Laura Theobald, Jennifer Pahne-Zeppenfeld, Subramanya Hegde, Yoo-Jin Kim, Rainer Maria Bohle, Ingolf Juhasz-Böss, Erich-Franz Solomayer and Sigrun Smola DOI: 10.1158/0008-5472.CAN-18-1913 Abstract Persistent infection with high-risk human papillomavirus (HPV) is a prerequisite for the development of cervical cancer. HPV-transformed cells actively instruct their microenvironment, promoting chronic inflammation and cancer progression.
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