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Barend M E on Muck Rack

Barend M E

As seen in: Oxford University Press (OUP), Biomed Central (BMC), Genome Medicine
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Articles

Identification of a PRDM1-regulated T cell network to regulate atherosclerotic plaque inflammation - Genome Medicine

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By Dos Santos, Barend M E, van der Vorst, Emiel P. C
| Genome Medicine Verified
References Libby P, Ridker PM, Hansson GK. Progress and challenges in translating the biology of atherosclerosis. Nature. 2011;473(7347):317–25. Article CAS PubMed Google Scholar Shah PK. Mechanisms of plaque vulnerability and rupture. J Am Coll Cardiol. 2003;41(4, Supplement):S15-22. Article Google Scholar Bentzon JF, Otsuka F, Virmani R, Falk E. Mechanisms of plaque formation and rupture. Circ Res. 2014;114(12):1852–66. Article CAS PubMed Google Scholar Tabas I, Lichtman AH.
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Identification of a PRDM1-regulated T cell network to regulate atherosclerotic plaque inflammation

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By Dos Santos, Barend M E, van der Vorst, Emiel P. C
| gla.ac.uk
Jin, H. et al. (2025) Identification of a PRDM1-regulated T cell network to regulate atherosclerotic plaque inflammation. Genome Medicine, 17(1), 109. (doi: 10.1186/s13073-025-01541-6) (PMID:41039608) (PMCID:PMC12490039) Text 370474.pdf - Published Version Available under License Creative Commons Attribution. 2MB Abstract Background: Inflammation is a key driver of atherosclerosis, yet the mechanisms sustaining inflammation in human plaques remain poorly understood.
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Genome-scale metabolic network of human carotid plaque reveals the pivotal role of glutamine/glutamate metabolism in macrophage modulating plaque inflammation and vulnerability | Cardiovascular Diabetology | Full Text

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By Barend M E, Erik A L, RWTH Aachen
| Biomed Central (BMC)
Carotid atherosclerotic plaque tissue samples from the MaasHPS cohort (n = 24) were obtained from symptomatic patients who had experienced a transient ischemic attack or minor stroke resulting from carotid stenosis after which they had undergone carotid endarterectomy (CEA) surgery within 2–14 days following the onset of neurological symptoms. Detailed clinical characteristics of the patients have been previously documented and are available in Table S1 [17, 18].
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