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Barry McCormick

As seen in: Wiley Online Library, The Journal of Immunology
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Articles

ARAP3 protects from excessive formylated peptide‐induced microvascular leakage by acting on endothelial cells and neutrophils

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By Julia Chu, Barry McCormick, Kruthika Sundaram, Gareth Hardisty
| Wiley Online Library Verified
Introduction Vascular permeability is temporarily heightened during the normal inflammatory response to permit protein-rich plasma to enter the interstitial space and promote the timely removal of threats. Many pathological situations are characterised by excessive inflammation-associated vascular leakage.
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A Negative Feedback Loop Regulates Integrin Inactivation and Promotes Neutrophil Recruitment to Inflammatory Sites

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By Barry McCormick, Helen Craig, Julia Chu, Leo M. Carlin
| The Journal of Immunology Verified
Open Access Barry McCormick, Helen E. Craig, Julia Y. Chu, Leo M. Carlin, Marta Canel, Florian Wollweber, Matilda Toivakka, Melina Michael, Anne L. Astier, Laura Norton, Johanna Lilja, Jennifer M. Felton, Takehiko Sasaki, Johanna Ivaska, Ingeborg Hers, Ian Dransfield, Adriano G. Rossi and Sonja Vermeren J Immunol September 15, 2019, 203 (6) 1579-1588; DOI: https://doi.org/10.4049/jimmunol.1900443 Key Points A negative feedback loop, integrin–PI3K–ARAP3–integrin, controls integrin inactivation.
Open in Who Shared Issue with article?

A Negative Feedback Loop Regulates Integrin Inactivation and Promotes Neutrophil Recruitment to Inflammatory Sites

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By Barry McCormick, Helen Craig, Julia Chu, Leo M. Carlin
| The Journal of Immunology Verified
Open Access Barry McCormick, Helen E. Craig, Julia Y. Chu, Leo M. Carlin, Marta Canel, Florian Wollweber, Matilda Toivakka, Melina Michael, Anne L. Astier, Laura Norton, Johanna Lilja, Jennifer M. Felton, Takehiko Sasaki, Johanna Ivaska, Ingeborg Hers, Ian Dransfield, Adriano G. Rossi and Sonja Vermeren J Immunol August 19, 2019, ji1900443; DOI: https://doi.org/10.4049/jimmunol.1900443 Key Points A negative feedback loop, integrin–PI3K–ARAP3–integrin, controls integrin inactivation.
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