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Deletions of CDKN2A and MTAP Detected by Copy-Number Variation Array Are Associated with Loss of p16 and MTAP Protein in Pleural Mesothelioma
1. Introduction Pleural mesothelioma (PM) is an aggressive neoplasm arising from the mesothelial cells lining the pleural cavity and is strongly associated with previous exposure to asbestos [1]. The algorithm for diagnosis of malignant PM requires histomorphologic evidence of invasion and immunohistochemical evidence of the mesothelial nature of the tumor [2].
Glucocorticoids are differentially synthesized along the murine and human respiratory tree
ORIGINAL ARTICLE This article has been accepted for publication and undergone full peer review but has not been through the copyediting, typesetting, pagination and proofreading process, which may lead to differences between this version and the Version of Record. Please cite this article as doi:10.1111/all.15765. Abstract Background Synthetic glucocorticoids (GC) are effective in the treatment of inflammatory diseases of the lung. However, long-term use leads to severe side effects.
Swiss Medical Weekly - Occupations and geographical distribution of mesothelioma in Switzerland 1989-2018 - record linkage of an asbestos-exposed population with the Swiss National Cohort
Original article a Division of Occupational and Environmental Medicine, University of Zurich, Switzerland b University Hospital Zurich, Zurich, Switzerland c Silag - Swiss Laboratory for Particle Analysis in Tissues, Zurich, Switzerland d Institute of Pathology, Cantonal Hospital of Thurgau, Switzerland e Epidemiology, Biostatistics and Prevention Institute, University of Zurich, Switzerland * Shared first authorship Summary OBJECTIVE: We investigated the possibility of linking the data of...
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